CONDITIONS BEING TREATED WITH HYPERBARIC OXYGEN THERAPY

Following is a list of conditions currently being treated with HBOT worldwide.  Most are considered experimental and controversial by the mainstream medical community, with only testimonials and anecdotal evidence for benefit. Medical insurance in the United States and elsewhere will often refuse payment.

(* Conditions marked with an asterisk below are sometimes reimbursed for a short time by Medicare and other medical insurance in the U.S., but only if strictly defined diagnostic criteria are met.)

Emergency Indications

 

 Air or Gas embolism* (in divers, but can also occur during bypass surgery)

 Bends in divers (Decompression sickness)*

 Burns* (thermal burns)

 Carbon monoxide poisoning* (Insurance usually stops paying before all brain injury has fully improved)  

 Cyanide Poisoning*

 Cerebral edema

 Closed head injuries (traumatic brain injury)

 Crisis of sickle cell anemia

 Exceptional Blood Loss Anemia*

 Blast injury

 Gas gangrene*

 Hydrogen sulfide poisoning

 Near-drowning

 Near-electrocution

 Near-hanging

 Peyote poisoning

 Severed limbs

 Smoke inhalation

 Ileus

Stroke (cerebral infarct, with benefit even long after the event)

 Specific Neurologic Indications

 

Autism

Air embolism*

   a. Decompression induced (divers)

   b. Iatrogenic (bypass surgery, injected, etc.)

Cerebral edema

   a. Toxic encephalopathy

   b. Vascular compromise

   c. Traumatic

Cerebral Palsy (CP)

Spinal cord contusion

   a. Physiological transection

   b. Partial motor or sensory loss

Early organic brain syndrome

    a. Small vessel disease

    b. Multiple small infarcts

Fetal alcohol syndrome

Stroke (acute and chronic residuals)

Vegetative coma (acute and prolonged)

    a. Closed head injury

    b. Hypoxic encephalopathy (suffocation, drowning, strangulation,

          cardiac arrest, near hanging, near choking, lightening strikes, etc.)

Multiple sclerosis

    a. Acute

    b. Relapsing/ remitting

    c. Chronic progressive

Brain Ddysfunction following Bypass Surgery and Angioplasty

Cranial nerve syndromes

    a. Trigeminal neuralgia

    b. Optic neuritis

    c. Vestibular disorders (vertigo, tinnitus, Meniere's syndrome)

    d. Sudden deafness

    e. Brain stem syndromes (ischemia, infarct)

    f. Retinal artery occlusion

    g Acoustic trauma

Peripheral neuropathy

     a. Charcot Marie's tooth disease

     b. Radiation myelitis

Traumatic Brain Injury

Near Drowning

Sympathetic Reflex Dystrophy

Orthopedic Indications

 

Crush injuries* (vascular compromise)

Soft tissue swelling

     a. Traumatic

    b. Cellulitis* (infection/mixed flora)

compartment syndrome*

Acute necrotizing fasciitis ( so-called "flesh-eating bacteria")*

Necrotizing soft tissue infections*

Clostridial myonecrosis* (gas gangrene)

Severed limbs and digits*

Acute and chronic osteomyelitis* (bone infection)

Bone grafting

Fracture healing and nonunion*

Aseptic necrosis

Tendon and ligament injuries, post-surgical repair

Delayed wound healing

Stump infections (following amputation)

Edema under cast

Sports injuries

Miscellaneous Indications

 

Peripheral vascular ulcer*

      a. Arterial (atherosclerosis)

      b. Decubitus (bed sores)

      c. Neuropathy related (diabetes)

      d. Venous (deep vein thrombosis complication)

      e. Diabetic

Gangrene (wet and dry, atherosclerosis, more common in diabetes)

Compromised Skin Flaps and Grafts*

Autism

Abscesses* (intra-abdominal and intracranial)

Buerger's disease

Biliary atresia (after surgical correction)

Carbon tetrachloride poisoning

Fibromyalgia

Frostbite

Diabetic retinopathy

Glaucoma with visual field loss

Diabetic ulcers*

Gulf War Syndrome

Retinal artery occlusion

Retinal vein thrombosis

Lepromatous leprosy

Lyme disease

Melaney ulcer* (flesh-eating bacteria)

Meningitis

Migraine

Osteoradionecrosis* (post radiation damage)

Pancreatitis

Pneumatosis cystoides intestinalis

Pseudomembranous colitis

Plastic surgery (speed healing, reduce scar and infection)

Rheumatoid arthritis (acute)

Brown Recluse Spider Bite

Scleroderma

Sickle cell crisis and hematuria

Peptic ulcer

Plastic Surgery, speeds healing with less scar

Myocardial infarction

Slow healing wounds

Potentiate antibiotic therapy

Post-cardiotomy and low output heart failure

Radiation cystitis and enteritis*

Refractory mycoses* (fungal infections)

Suturing of Severed Limbs*

Chronic Fatigue Syndrome

Cerebral palsy

Post-polio syndrome

Cirrhosis

Immune System Enhancement

Crohn's disease

Ulcerative colitis

ALS

Parkinson's Syndrome

Research Report of Hyperbaric Oxygen Benefit for Stroke Victims

Published clinical studies describing results from treating stroke patients with hyperbaric oxygen, performed at 11 different hyperbaric centers, were analyzed for benefit (a total of 265 patients). The cumulative amount of hyperbaric oxygen therapy (DHBOT) was calculated by multiplying chamber oxygen pressure (ATA) times the duration of each HBOT (in hours), times the total number of hyperbaric treatments. Efficacy of HBOT (EfHBOT) was computed from the number of patients in each study who showed significant clinical improvement of their neurologic status as a result of HBOT (percentage of the total number of patients who improved). The amount of benefit was compared with the total amount of HBOT. Analysis showed that benefit increased progressively as more treatments were given, as graphically depicted below.

 

From the graphic plot above it can be seen that average improvement increased with each treatment and that 30 one-hour HBOT treatments resulted in progressive benefit. For many patients that benefit continued to increase with additional therapy and 30 treatments is not usually considered adequate for full benefit.

Reference below linked to publication and abstract in the National Library of Medicine, NIH, Database

Rogatsky GG, Shifrin EG, Mayevsky A. Optimal dosing as a necessary condition for the efficacy of hyperbaric oxygen therapy in acute ischemic stroke: a critical review. Neurol Res. 2003 Jan;25(1):95-8

                                                      

FORT LAUDERDALE, FLA. — Hyperbaric oxygen therapy improves cognitive and social function in children with chronic brain injury, according to a study presented at a symposium on hyperbaric oxygen therapy.

Daily living, socialization, communication, and motor skills significantly improved for 21 children treated with hyperbaric oxygen therapy (HBOT), compared with 21 brain-injured patients who received standard therapy. Researchers included a third group of 21 healthy children to control for normal growth and development, reported Dr. Charles J. Golden at the symposium sponsored by the Ocean Hyperbaric Neurologic Center.

Participants were assessed more than 1 year after onset of their brain injury. The majority had cerebral palsy. The average age was 4.5 years (range, 12 months to 18 years), said Dr. Golden, professor of psychology and director, Neuropsychology Assessment Center, Nova Southeastern University, Fort Lauderdale, Fla.

Average functioning level was close to two standard deviations below average—“so this was a very low functioning group,” he added.

Mild changes in some areas but no changes in the cerebellum were noted after 35 HBOT sessions, compared with baseline, Dr. Golden said. “This is not unexpected. These children had injuries high up in the brain.”

“Interestingly, you can predict reasonably well who will be a responder based on response over the first 35 treatments,” he said. “Some people are just nonresponders—you can give them 200 treatments, and they will not respond. Others are marvelous responders who respond well and right away.”

After a second round of 35 HBOT treatments, “there was a much greater effect on blood flow—so it seems to be a time-based effect,” Dr. Golden said.

The HBOT group made major changes in all areas that were greater than either the normal or standard therapy control groups.

“This is a group who is at the end—they have failed multiple therapies. And still we have about 70% who respond [to HBOT],” said Dr. Golden. “The plasticity of the brain may be much greater than we imagined. HBOT may stimulate ability of the brain to reorganize itself.”

Dr. Golden and his associates used the Vineland Adaptive Behavior Scales to rate basic adaptive, motor, and cognitive abilities “This can be used without a child having to perform for us, which is challenging with cerebral palsy,” he said.

They assessed blood flow changes with a series of three single-photon emission computed tomography (SPECT) scans before, during, and after HBOT treatment. They assessed the cerebellum, pons, right and left hemisphere subcortical areas, and the cortical region.

"Improvements in motor functions [from HBOT] allowed them to do things they could not do at the beginning of the study."

 

Damian McNamara, Hyperbaric Oxygen Therapy Helps Children Who Have Chronic Brain Injury. Family Practice News, Volume 36, Issue 19, Page 49 (01 October 2006)